peroxidase conjugated anti mouse igg antibody (Vector Laboratories)
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Vector Laboratories
peroxidase conjugated anti mouse igg antibody
Peroxidase Conjugated Anti Mouse Igg Antibody, supplied by Vector Laboratories, used in various techniques. Bioz Stars score: 92/100, based on 20 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/WB-2000-%2E8/WestVision+Peroxidase+Polymer+Anti-Mouse+IgG+for+Western+Blot+Detection/pm42882508-97-17-21
Average 92 stars, based on 20 article reviews
Peroxidase Conjugated Anti Mouse Igg Antibody, supplied by Vector Laboratories, used in various techniques. Bioz Stars score: 92/100, based on 20 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/WB-2000-%2E8/WestVision+Peroxidase+Polymer+Anti-Mouse+IgG+for+Western+Blot+Detection/pm42882508-97-17-21
Average 92 stars, based on 20 article reviews
peroxidase conjugated anti mouse igg antibody - by Bioz Stars,
2026-11
92/100 stars
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Western Blot:Article Title: SOX21 suppresses GBM growth by repressing AP-1 regulated target genes Article Snippet: Subsequently, proteins were transferred to a nitrocellulose blotting membrane (Amersham 0.45 μm, 10600016) and blocked in 5% milk in PBS/0.1% Tween (Sigma, P1379) followed by primary antibody incubation overnight in a cold room. .. Anti-mouse or anti-rabbit horseradish peroxidase-coupled secondary antibody (Vector Article Title: Low-Dose FK-506 Contributes to Tubulointerstitial Fibrosis Through Epithelial-Stromal Interactions Article Snippet: The membrane was subjected to incubation at 4 °C for 18 h with various antibody-targeting proteins, including αSMA (Sigma-Aldrich, St. Louis, MO, USA; #A5228), vimentin (Santa Cruz Biotechnology, Santa Cruz, CA, USA; #sc-6260), fibronectin (ABclonal, Woburn, MA, USA; #A12932), N-cadherin (Cell Signaling Technology, Beverly, MA, USA; #4061), E-cadherin (Santa Cruz Biotechnology, #sc-7870), PARP1 (Cell Signaling Technology, #9542), LC3 (Cell Signaling Technology, #12741), GPX4 (Santa Cruz Biotechnology, #sc-166570), P27 (Cell Signaling Technology, #2552), and b-actin (Santa Cruz Biotechnology; #sc-47778). .. Subsequently, peroxidase-conjugated anti-rabbit (Vector Laboratories, Burlingame, CA, USA; #WB-1000) and Article Title: Activation of Yes-Associated Protein Is Indispensable for Transformation of Kidney Fibroblasts into Myofibroblasts during Repeated Administration of Cisplatin Article Snippet: The membrane was subjected to incubation at 4 °C for 18 h with various antibody-targeting proteins, including α-SMA (Sigma-Aldrich; product no. A5228), vimentin (Santa Cruz Biotechnology, Santa Cruz, CA, USA; product no. sc-6260), fibronectin (ABclonal, Woburn, MA, USA; product no. A12932), p21 (Santa Cruz Biotechnology; product no. sc-6246), JNK (Cell Signaling Technology, Beverly, MA, USA; product no. 9252), phosphorylated JNK (p-JNK; Cell Signaling Technology; product no. 9251), YAP (Cell Signaling Technology; product no. 4912), phosphorylated YAP (p-YAP; Cell Signaling Technology; product no. 13008), phosphorylated monopolar spindle one binder kinase activator-like 1 (p-MOB1; Cell Signaling Technology; product no. 8699S), phosphorylated macrophage stimulating protein 1/2 (p-MST1/2; Proteintech; product no. 80093-1-RR), connective tissue growth factor (CTGF; Abcam, Cambridege, MA, USA; product no. ab6992), and β-actin (Santa Cruz Biotechnology; product no. sc-47778). .. Subsequently, peroxidase-conjugated anti-rabbit (Vector Laboratories, Burlingame, CA, USA; product no. WB-1000) and Article Title: PARP1 Deficiency Attenuates Cisplatin Nephrotoxicity and Renal COX-2/PGE2 Response. Article Snippet: Equal amounts of protein (20–40 μg) were separated by SDS– polyacrylamide gel electrophoresis, transferred to polyvinylidene fluoride membranes, and incubated overnight at 4 ◦C with primary antibodies against phosphorylated NFκB p65 (p-p65; ABclonal Technology, Woburn, MA, USA; no. AP0123; 1:2500 dilution), total NF-κB p65 (t-p65; ABclonal Technology; no. A22331; 1:2500 dilution), COX-2 (ABclonal Technology; no. A3560; 1:2500 dilution), and β-actin (Santa Cruz Biotechnology, Santa Cruz, CA, USA; no. sc-47778; 1:5000 dilution). .. After incubation with peroxidaseconjugated anti-rabbit IgG antibody (Vector Laboratories, Newark, CA, USA; no. WB-1000; 1:5000 dilution) or Article Title: Inhibition of neuronal peroxisome proliferator-activated receptor-γ attenuates motor function improvement after spinal cord injury in rats. Article Snippet: Edited by: Vidita Vaidya Abstract Traumatic spinal cord injury (SCI) causes secondary damage in injured and adjacent regions due to temporal deprivation of oxygen and energy supply.. Peroxisome proliferator-activated receptor γ (PPARγ) is known to regulate cell survival mechanisms such as hypoxia, oxidative stress, inflammation and energy homeostasis in various tissues.. Thus, PPARγ has the potential to show neuroprotective properties. Article Title: Mammalian Target of Rapamycin Inhibition Decreases Angiotensin II-Induced Steroidogenesis in HAC15 Human Adrenocortical Carcinoma Cells. Article Snippet: Background.. Mammalian target of rapamycin (mTOR) inhibitors suppress adrenal cortical carcinoma cell proliferation and cortisol production; the relationship between mTOR and aldosterone production has not been examined.. Methods. Article Title: Loss of SAV1 in Kidney Proximal Tubule Induces Maladaptive Repair after Ischemia and Reperfusion Injury Article Snippet: The membranes were incubated with antibodies against SAV1 (Cell Signaling Technology; catalog no. 13301; 1:5000 dilution), phosphorylated MOB1 (p-MOB1; Cell Signaling Technology; catalog no. 8699S; 1:5000 dilution), phosphorylated YAP (p-YAP; Cell Signaling Technology; catalog no. 13008; 1:5000 dilution), total YAP (t-YAP; Cell Signaling Technology; catalog no. 4912; 1:5000 dilution), PI3K (Cell Signaling Technology; catalog no. 4292; 1:2500 dilution), phosphorylated AKT (p-AKT; ABclonal Technology, Woburn, MA, USA; catalog no. AP0637; 1:2500 dilution), total AKT (t-AKT; Santa Cruz Biotechnology, Santa Cruz, CA, USA; catalog no. sc-8312; 1:2500 dilution), phosphorylated ERK (p-ERK; Cell Signaling Technology; catalog no. 4370; 1:5000 dilution), total ERK (t-ERK; Santa Cruz Biotechnology; catalog no. sc-93; 1:10,000 dilution), and β-actin (Santa Cruz Biotechnology; catalog no. sc-47778; 1:5000 dilution) overnight at 4 °C. .. After washing, peroxidase anti-rabbit IgG antibody (Vector Laboratories; catalog no. WB-1000; 1:5000 dilution) against SAV1, p-MOB1, p-YAP, t-YAP, PI3K, p-AKT, t-AKT, p-ERK, and t-ERK antibodies, and Incubation:Article Title: SOX21 suppresses GBM growth by repressing AP-1 regulated target genes Article Snippet: Subsequently, proteins were transferred to a nitrocellulose blotting membrane (Amersham 0.45 μm, 10600016) and blocked in 5% milk in PBS/0.1% Tween (Sigma, P1379) followed by primary antibody incubation overnight in a cold room. .. Anti-mouse or anti-rabbit horseradish peroxidase-coupled secondary antibody (Vector Article Title: PARP1 Deficiency Attenuates Cisplatin Nephrotoxicity and Renal COX-2/PGE2 Response. Article Snippet: Equal amounts of protein (20–40 μg) were separated by SDS– polyacrylamide gel electrophoresis, transferred to polyvinylidene fluoride membranes, and incubated overnight at 4 ◦C with primary antibodies against phosphorylated NFκB p65 (p-p65; ABclonal Technology, Woburn, MA, USA; no. AP0123; 1:2500 dilution), total NF-κB p65 (t-p65; ABclonal Technology; no. A22331; 1:2500 dilution), COX-2 (ABclonal Technology; no. A3560; 1:2500 dilution), and β-actin (Santa Cruz Biotechnology, Santa Cruz, CA, USA; no. sc-47778; 1:5000 dilution). .. After incubation with peroxidaseconjugated anti-rabbit IgG antibody (Vector Laboratories, Newark, CA, USA; no. WB-1000; 1:5000 dilution) or Article Title: Inhibition of neuronal peroxisome proliferator-activated receptor-γ attenuates motor function improvement after spinal cord injury in rats. Article Snippet: Edited by: Vidita Vaidya Abstract Traumatic spinal cord injury (SCI) causes secondary damage in injured and adjacent regions due to temporal deprivation of oxygen and energy supply.. Peroxisome proliferator-activated receptor γ (PPARγ) is known to regulate cell survival mechanisms such as hypoxia, oxidative stress, inflammation and energy homeostasis in various tissues.. Thus, PPARγ has the potential to show neuroprotective properties. other:Article Title: Metformin and thymoquinone co-treatment enhance 5-fluorouracil cytotoxicity by suppressing the PI3K/mTOR/HIF1α pathway and increasing oxidative stress in colon cancer cells. Article Snippet: Funding information The Deputyship for Research & Innovation, Ministry of Education in Saudi Arabia, Grant/Award Number: IFP22UQU4320045DSR101 Abstract This study investigated the chemotherapeutic effects of 5-fluorouracil (5-FU), metformin (Met), and/or thymoquinone (TQ) single/dual/triple therapies in the HT29, SW480 and SW620 colon cancer (CRC) cell lines.. Cell cycle/ apoptosis were measured by flow cytometry.. The gene and protein expression of apoptosis (PCNA/survivin/BAX/Cytochrome-C/Caspase-3) and cell cycle (CCND1/CCND3/p21/p27) molecules, the PI3K/mTOR/HIF1α oncogenic pathway, and glycolysis regulatory enzymes were measured by quantitativePCR and Western blot. Imaging:Article Title: PARP1 Deficiency Attenuates Cisplatin Nephrotoxicity and Renal COX-2/PGE2 Response. Article Snippet: Equal amounts of protein (20–40 μg) were separated by SDS– polyacrylamide gel electrophoresis, transferred to polyvinylidene fluoride membranes, and incubated overnight at 4 ◦C with primary antibodies against phosphorylated NFκB p65 (p-p65; ABclonal Technology, Woburn, MA, USA; no. AP0123; 1:2500 dilution), total NF-κB p65 (t-p65; ABclonal Technology; no. A22331; 1:2500 dilution), COX-2 (ABclonal Technology; no. A3560; 1:2500 dilution), and β-actin (Santa Cruz Biotechnology, Santa Cruz, CA, USA; no. sc-47778; 1:5000 dilution). .. After incubation with peroxidaseconjugated anti-rabbit IgG antibody (Vector Laboratories, Newark, CA, USA; no. WB-1000; 1:5000 dilution) or Membrane:Article Title: Inhibition of neuronal peroxisome proliferator-activated receptor-γ attenuates motor function improvement after spinal cord injury in rats. Article Snippet: Edited by: Vidita Vaidya Abstract Traumatic spinal cord injury (SCI) causes secondary damage in injured and adjacent regions due to temporal deprivation of oxygen and energy supply.. Peroxisome proliferator-activated receptor γ (PPARγ) is known to regulate cell survival mechanisms such as hypoxia, oxidative stress, inflammation and energy homeostasis in various tissues.. Thus, PPARγ has the potential to show neuroprotective properties. Saline:Article Title: Inhibition of neuronal peroxisome proliferator-activated receptor-γ attenuates motor function improvement after spinal cord injury in rats. Article Snippet: Edited by: Vidita Vaidya Abstract Traumatic spinal cord injury (SCI) causes secondary damage in injured and adjacent regions due to temporal deprivation of oxygen and energy supply.. Peroxisome proliferator-activated receptor γ (PPARγ) is known to regulate cell survival mechanisms such as hypoxia, oxidative stress, inflammation and energy homeostasis in various tissues.. Thus, PPARγ has the potential to show neuroprotective properties. Polymer:Article Title: Inhibition of neuronal peroxisome proliferator-activated receptor-γ attenuates motor function improvement after spinal cord injury in rats. Article Snippet: Edited by: Vidita Vaidya Abstract Traumatic spinal cord injury (SCI) causes secondary damage in injured and adjacent regions due to temporal deprivation of oxygen and energy supply.. Peroxisome proliferator-activated receptor γ (PPARγ) is known to regulate cell survival mechanisms such as hypoxia, oxidative stress, inflammation and energy homeostasis in various tissues.. Thus, PPARγ has the potential to show neuroprotective properties. |